c-Ets1 inhibits the interaction of NF-κB and CREB, and downregulates IL-1β-induced MUC5AC overproduction during airway inflammation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
http://www.nature.com/articles/mi201167.pdf
Reference38 articles.
1. Song, K.S., Kim, H.J. & Lee, J.G. Rgulator of G-protein signaling 4 suppresses LPS-induced MUC5AC overproduction in the airway. Am. J. Respir. Cell Mol. Biol. 41, 40–49 (2009).
2. Song, K.S., Choi, Y.H., Kim, J.M., Lee, H., Lee, T.J. & Yoon, J.H. Suppression of prostaglandin E2-induced MUC5AC overproduction by RGS4 in the airway. Am. J. Physiol. Lung Cell Mol. Physiol. 296, L684–L692 (2009).
3. Song, K.S., Kim, K., Chung, K.C., Seol, J.H. & Yoon, J.H. Interaction of SOCS3 with NonO attenuates IL-1beta-dependent MUC8 gene expression. Biochem. Biophys. Res. Commun. 377, 946–951 (2008).
4. Fujisawa, T., Velichko, S., Thai, P., Hung, L.Y., Huang, F. & Wu, R. Regulation of airway MUC5AC expression by IL-1beta and IL-17A; the NF-kappaB paradigm. J. Immunol. 183, 6236–6243 (2009).
5. Song, K.S. et al. Interleukin-1 beta and tumor necrosis factor-alpha induce MUC5AC overexpression through a mechanism involving ERK/p38 mitogen-activated protein kinases-MSK1-CREB activation in human airway epithelial cells. J. Biol. Chem. 278, 23243–23250 (2003).
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