CAMK2γ in intestinal epithelial cells modulates colitis-associated colorectal carcinogenesis via enhancing STAT3 activation-Reference-Cited by-同舟云学术

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CAMK2γ in intestinal epithelial cells modulates colitis-associated colorectal carcinogenesis via enhancing STAT3 activation

Published:2017-03-20 Issue:28 Volume:36 Page:4060-4071
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Ma X,Meng Z,Jin L,Xiao Z,Wang X,Tsark W M,Ding L,Gu Y,Zhang J,Kim B,He M,Gan X,Shively J E,Yu H,Xu R,Huang W

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

https://www.nature.com/articles/onc201716.pdf

Reference67 articles.

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2. Trinchieri G . Cancer and inflammation: an old intuition with rapidly evolving new concepts. Annu Rev Immunol 2012; 30: 677–706.

3. Polk DB, Peek RM Jr . Helicobacter pylori: gastric cancer and beyond. Nat Rev Cancer 2010; 10: 403–414.

4. Wotherspoon AC . A critical review of the effect of Helicobacter pylori eradication on gastric MALT lymphoma. Curr Gastroenterol Rep 2000; 2: 494–498.

5. Arzumanyan A, Reis HM, Feitelson MA . Pathogenic mechanisms in HBV- and HCV-associated hepatocellular carcinoma. Nat Rev Cancer 2013; 13: 123–135.

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