Overactive mitochondrial DNA replication disrupts perinatal cardiac maturation
Author:
Funder
Sigrid Juséliuksen Säätiö
Academy of Finland
Maud Kuistilan Muistosäätiö
Päivikki ja Sakari Sohlbergin Säätiö
Biomedicum Helsinki-säätiö
Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41467-024-52164-1.pdf
Reference45 articles.
1. Gorman, G. S. et al. Mitochondrial diseases. Nat. Rev. Dis. Prim. 2, 16080 (2016).
2. Ikeda, M. et al. Overexpression of TFAM or twinkle increases mtDNA copy number and facilitates cardioprotection associated with limited mitochondrial oxidative stress. PLoS ONE 10, e0119687 (2015).
3. Pohjoismäki, J. L. et al. Overexpression of Twinkle-helicase protects cardiomyocytes from genotoxic stress caused by reactive oxygen species. Proc. Natl Acad. Sci. USA. 110, 19408–19413 (2013).
4. Tanaka, A. et al. The overexpression of twinkle helicase ameliorates the progression of cardiac fibrosis and heart failure in pressure overload model in mice. PLoS ONE 8, e67642 (2013).
5. Inoue, T. et al. Twinkle overexpression prevents cardiac rupture after myocardial infarction by alleviating impaired mitochondrial biogenesis. Am. J. Physiol. - Heart Circ. Physiol. 311, 509–519 (2016).
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