Hepatotoxicity profile of single agent arsenic trioxide in the treatment of newly diagnosed acute promyelocytic leukemia, its impact on clinical outcome and the effect of genetic polymorphisms on the incidence of hepatotoxicity-Reference-Cited by-同舟云学术

Hepatotoxicity profile of single agent arsenic trioxide in the treatment of newly diagnosed acute promyelocytic leukemia, its impact on clinical outcome and the effect of genetic polymorphisms on the incidence of hepatotoxicity

Published:2006-03-09 Issue:5 Volume:20 Page:881-883
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Mathews V,Desire S,George B,Lakshmi K M,Rao J G,Viswabandya A,Bajel A,Srivastava V M,Srivastava A,Chandy M

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/2404165.pdf

Reference9 articles.

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3. Brouwer OF, Onkenhout W, Edelbroek PM, de Kom JF, de Wolff FA, Peters AC . Increased neurotoxicity of arsenic in MTHFR deficiency. Clin Neurol Neurosurg 1992; 94: 307–310.

4. Chiou HY, Hsueh YM, Hsieh LL, Hsu LI, Hsu YH, Hsieh FI et al. Arsenic methylation capacity, body retention and null genotypes of glutathione S-transferases M1 and T1 among current arsenic exposed residents of Taiwan. Mutat Res 1997; 386: 197–207.

5. Coles BF, Morel F, Rauch C, Huber WW, Yang M, Teitel CH et al. Effect of polymorphism in the human glutathione S-transferase A1 promoter on hepatic GSTA1 and GSTA2 expression. Pharmacogenetics 2001; 11: 663–669.

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