Involvement of antibody production quantitative trait loci in the susceptibility to pristane-induced arthritis in the mouse

Author:

Jensen J R,Peters L C,Borrego A,Ribeiro O G,Cabrera W H K,Starobinas N,Siqueira M,Ibañez O C M,De Franco M

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics,Immunology

Reference31 articles.

1. Cooper GS, Stroehla BC . The epidemiology of autoimmune diseases. Autoimmun Rev 2003; 2: 119–125.

2. Jawaheer D, Thomson W, MacGregor AJ, Carthy D, Davidson J, Dyer PA et al. Homozygosity for the HLA-Dr shared epitope contributes the highest risk for rheumatoid arthritis concordance in identical twins. Arthritis Rheum 1994; 37: 681–686.

3. John S, Marlow A, Hajeer A, Ollier WE, Silman AJ, Worthington J . Linkage and association studies of the natural resistance associated macrophage protein-1 (Nramp1) locus in rheumatoid arthritis. J Rheumatol 1997; 24: 452–457.

4. Potter M, Wax JS . Genetics of susceptibility to pristane-induced plasmocytomas in Balb/cJ with a brief description of pristane-induced arthritis. J Immunol 1981; 127: 1591–1595.

5. Stasiuk L, Ghoraisian M, Elson CJ, Thompson SJ . Pristane-induced arthritis is CD4+ T-cell dependent. Immunology 1997; 90: 81–86.

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