Far-infrared radiation protects viability in a cell model of Spinocerebellar Ataxia by preventing polyQ protein accumulation and improving mitochondrial function-Reference-Cited by-同舟云学术

Far-infrared radiation protects viability in a cell model of Spinocerebellar Ataxia by preventing polyQ protein accumulation and improving mitochondrial function

Published:2016-07-29 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Chang Jui-Chih,Wu Shey-Lin,Hoel Fredrik,Cheng Yu-Shan,Liu Ko-Hung,Hsieh Mingli,Hoel August,Tronstad Karl Johan,Yan Kuo-Chia,Hsieh Ching-Liang,Lin Wei-Yong,Kuo Shou-Jen,Su Shih-Li,Liu Chin-San

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep30436.pdf

Reference48 articles.

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3. Costa Mdo, C. & Paulson, H. L. Toward understanding Machado-Joseph disease. Progress in neurobiology 97, 239–257, doi: 10.1016/j.pneurobio.2011.11.006 (2012).

4. Bhat, A. H. et al. Oxidative stress, mitochondrial dysfunction and neurodegenerative diseases; a mechanistic insight. Biomed Pharmacother 74, 101–110, doi: 10.1016/j.biopha.2015.07.025 (2015).

5. Yan, M. H., Wang, X. & Zhu, X. Mitochondrial defects and oxidative stress in Alzheimer disease and Parkinson disease. Free radical biology & medicine 62, 90–101, doi: 10.1016/j.freeradbiomed.2012.11.014 (2013).

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