Reply to Matters Arising: Is congenital anosmia protective for Parkinson’s disease triggered by pathogenic entrance through the nose?
Author:
Funder
Vetenskapsrådet
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Neurology
Link
https://www.nature.com/articles/s41531-023-00539-4.pdf
Reference13 articles.
1. Arshamian, A., Iravani, B. & Lundström, J. N. Is congenital anosmia protective for Parkinson’s disease triggered by pathogenic entrance through the nose? NPJ Parkinsons Dis. 8, 152 (2022).
2. Rey, N. L., Wesson, D. W. & Brundin, P. The olfactory bulb as the entry site for prion-like propagation in neurodegenerative diseases. Neurobiol. Dis. 109, 226–248 (2018).
3. Wenning, G. K. et al. Olfactory function in atypical parkinsonian syndromes. Acta Neurol. Scand. 91, 247–250 (1995).
4. Hawkes, C. H., Del Tredici, K. & Braak, H. Parkinson’s disease: a dual-hit hypothesis. Neuropathol. Appl. Neurobiol. 33, 599–614 (2007).
5. Borghammer, P. et al. A postmortem study suggests a revision of the dual-hit hypothesis of Parkinson’s disease. npj Parkinsons Dis. 8, 1–11 (2022).
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