mTOR/Raptor signaling is critical for skeletogenesis in mice through the regulation of Runx2 expression-Reference-Cited by-同舟云学术

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mTOR/Raptor signaling is critical for skeletogenesis in mice through the regulation of Runx2 expression

Published:2017-07-07 Issue:11 Volume:24 Page:1886-1899
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Dai Qinggang,Xu Zhan,Ma Xuhui,Niu Ningning,Zhou Siru,Xie Furong,Jiang Lingyong,Wang Jun,Zou Weiguo

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/cdd2017110.pdf

Reference39 articles.

1. Harada S, Rodan GA . Control of osteoblast function and regulation of bone mass. Nature 2003; 423: 349–355.

2. Guntur AR, Rosen CJ . The skeleton: a multi-functional complex organ. New insights into osteoblasts and their role in bone formation: the central role of PI3Kinase. J Endocrinol 2011; 211: 123–130.

3. Ducy P, Zhang R, Geoffroy V, Ridall AL, Karsenty G . Osf2/Cbfa1: a transcriptional activator of osteoblast differentiation. Cell 1997; 89: 747–754.

4. Otto F, Thornell AP, Crompton T, Denzel A, Gilmour KC, Rosewell IR et al. Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development. Cell 1997; 89: 765–771.

5. Komori T, Yagi H, Nomura S, Yamaguchi A, Sasaki K, Deguchi K et al. Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts. Cell 1997; 89: 755–764.

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