The reversible epigenetic silencing of BRM: implications for clinical targeted therapy-Reference-Cited by-同舟云学术

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The reversible epigenetic silencing of BRM: implications for clinical targeted therapy

Published:2007-06-04 Issue:49 Volume:26 Page:7058-7066
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Glaros S,Cirrincione G M,Muchardt C,Kleer C G,Michael C W,Reisman D

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/1210514.pdf

Reference20 articles.

1. Bourachot B, Yaniv M, Muchardt C . (2003). Growth inhibition by the mammalian SWI-SNF subunit Brm is regulated by acetylation. EMBO J 22: 6505–6515.

2. Coisy-Quivy M, Disson O, Roure V, Muchardt C, Blanchard JM, Dantonel JC . (2006). Role for brm in cell growth control. Cancer Res 66: 5069–5076.

3. Fukuoka J, Fujii T, Shih JH, Dracheva T, Meerzaman D, Player A et al. (2004). Chromatin remodeling factors and BRM/BRG1 expression as prognostic indicators in non-small cell lung cancer. Clin Cancer Res 10: 4314–4324.

4. Khavari PA, Peterson CL, Tamkun JW, Mendel DB, Crabtree GR . (1993). BRG1 contains a conserved domain of the SWI2/SNF2 family necessary for normal mitotic growth and transcription. Nature 366: 170–174.

5. Klochendler-Yeivin A, Muchardt C, Yaniv M . (2002). SWI/SNF chromatin remodeling and cancer. Curr Opin Genet Dev 12: 73–79.

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