Mutation of p53 and consecutive selective drug resistance in B-CLL occurs as a consequence of prior DNA-damaging chemotherapy-Reference-Cited by-同舟云学术

Mutation of p53 and consecutive selective drug resistance in B-CLL occurs as a consequence of prior DNA-damaging chemotherapy

Published:2003-04 Issue:4 Volume:10 Page:477-484
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Sturm I,Bosanquet A G,Hermann S,Güner D,Dörken B,Daniel P T

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/4401194.pdf

Reference54 articles.

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2. Hemmati PG, Gillissen B, von Haefen C, Wendt J, Stärck L, Güner D, Dörken B and Daniel PT (2002) Adenovirus-mediated overexpression of p14ARF induces p53 and bax-independent apoptosis. Oncogene 21: 3149–3161

3. Friedrich K, Wieder T, von Haefen C, Radetzki S, Schulze-Osthoff K, Jänicke R, Dörken B and Daniel PT (2001) Overexpression of caspase-3 restores sensitivity for drug-induced apoptosis in breast cancer cells with acquired drug resistance. Oncogene 20: 2749–2460

4. von Haefen C, Wieder T, Gillissen B, Stärck L, Graupner V, Dörken B and Daniel PT (2002) Ceramide induces mitochondrial activation and apoptosis via a bax-dependent pathway in human carcinoma cells. Oncogene 21: 4009–4019

5. Wu GS and El-Deiry WS (1996) Apoptotic death of tumor cells correlates with chemosensitivity, independent of p53 or bcl-2. Clin. Cancer Res. 2: 623–633

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