Treatment of T1D via optimized expansion of antigen-specific Tregs induced by IL-2/anti-IL-2 monoclonal antibody complexes and peptide/MHC tetramers-Reference-Cited by-同舟云学术

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Treatment of T1D via optimized expansion of antigen-specific Tregs induced by IL-2/anti-IL-2 monoclonal antibody complexes and peptide/MHC tetramers

Published:2018-05-25 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Izquierdo Cristina,Ortiz Angela Zarama,Presa Maximiliano,Malo Sara,Montoya Anna,Garabatos Nahir,Mora Conchi,Verdaguer Joan,Stratmann Thomas

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-26161-6.pdf

Reference48 articles.

1. Anderson, M. S. & Bluestone, J. A. The NOD mouse: A model of immune dysregulation. Ann. Rev. Immunol. 23, 447–485 (2005).

2. Chen, Z., Herman, A. E., Matos, M., Mathis, D. & Benoist, C. Where CD4+CD25+ T reg cells impinge on autoimmune diabetes. J. Exp. Med. 202, 1387–1397 (2005).

3. Feuerer, M., Shen, Y., Littman, D. R., Benoist, C. & Mathis, D. How punctual ablation of regulatory T cells unleashes an autoimmune lesion within the pancreatic islets. Immunity. 31, 654–664 (2009).

4. Alard, P. et al. Deficiency in NOD antigen-presenting cell function may be responsible for suboptimal CD4+CD25+ T-cell-mediated regulation and type 1 diabetes development in NOD mice. Diabetes. 55, 2098–2105 (2006).

5. Wu, A. J., Hua, H., Munson, S. H. & McDevitt, H. O. Tumor necrosis factor-alpha regulation of CD4+CD25+ T cell levels in NOD mice. Proc. Natl. Acad. Sci 99, 12287–12292 (2002).

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