Computational analysis of the evolutionarily conserved Missing In Metastasis/Metastasis Suppressor 1 gene predicts novel interactions, regulatory regions and transcriptional control
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-40697-1.pdf
Reference99 articles.
1. Machesky, L. M. & Johnston, S. A. MIM: a multifunctional scaffold protein. J. Mol. Med. Berl. Ger. 85, 569–576 (2007).
2. Lee, Y.-G., Macoska, J. A., Korenchuk, S. & Pienta, K. J. MIM, a potential metastasis suppressor gene in bladder cancer. Neoplasia N. Y. N 4, 291–294 (2002).
3. Mattila, P. K. et al. Missing-in-metastasis and IRSp53 deform PI(4,5)P2-rich membranes by an inverse BAR domain-like mechanism. J. Cell Biol. 176, 953–964 (2007).
4. Yamagishi, A., Masuda, M., Ohki, T., Onishi, H. & Mochizuki, N. A novel actin bundling/filopodium-forming domain conserved in insulin receptor tyrosine kinase substrate p53 and missing in metastasis protein. J. Biol. Chem. 279, 14929–14936 (2004).
5. Mattila, P. K., Salminen, M., Yamashiro, T. & Lappalainen, P. Mouse MIM, a tissue-specific regulator of cytoskeletal dynamics, interacts with ATP-actin monomers through its C-terminal WH2 domain. J. Biol. Chem. 278, 8452–8459 (2003).
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. MTSS1 inhibits colorectal cancer metastasis by regulating the CXCR4/CXCL12 signaling axis;International Journal of Molecular Medicine;2021-02-26
2. Missing-in-Metastasis/Metastasis Suppressor 1 Regulates B Cell Receptor Signaling, B Cell Metabolic Potential, and T Cell-Independent Immune Responses;Frontiers in Immunology;2020-04-16
3. Missing-In-Metastasis / Metastasis Suppressor 1 regulates B cell receptor signaling, B cell metabolic potential and T cell-independent immune responses;2019-09-25
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