Differential impact of lipopolysaccharide defects caused by loss of RfaH in Yersinia pseudotuberculosis and Yersinia pestis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-11334-6.pdf
Reference49 articles.
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2. Barnes, P. D., Bergman, M. A., Mecsas, J. & Isberg, R. R. Yersinia pseudotuberculosis disseminates directly from a replicating bacterial pool in the intestine. J Exp Med 203, 1591–1601, doi: 10.1084/jem.20060905 (2006).
3. Cornelis, G. R. et al. The virulence plasmid of Yersinia, an antihost genome. Microbiol Mol Bio Rev 62, 1315–52 (1998).
4. Viboud, G. I. & Bliska, J. B. Yersinia outer proteins: Role in modulation of host cell signaling responses and pathogenesis. Annu Rev Microbiol 59, 69–89, doi: 10.1146/annurev.micro.59.030804.121320 (2005).
5. Viboud, G. I., So, S. S. K., Ryndak, M. B. & Bliska, J. B. Proinflammatory signalling stimulated by the type III translocation factor YopB is counteracted by multiple effectors in epithelial cells infected with Yersinia pseudotuberculosis. Mol Microbiol 47, 1305–1315, doi: 10.1046/j.1365-2958.2003.03350.x (2003).
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