LMK-235 suppresses osteoclastogenesis and promotes osteoblastogenesis by inhibiting HDAC4-Reference-Cited by-同舟云学术

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LMK-235 suppresses osteoclastogenesis and promotes osteoblastogenesis by inhibiting HDAC4

Published:2024-08-28 Issue:1 Volume:14 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Chen Chongwei,Li Yue,Feng Teng,Chen Xinping,Li Chengwei,Li Lu,Zhu Mengbo,Chang Yaqiong,Wang Shaowei

Funder

National Natural Science Foundation of China

Research Project Supported by Shanxi Scholarship Council of China

Natural Science Foundation for Young Scientists of Shanxi Province

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s41598-024-70814-8.pdf

Reference40 articles.

1. Loi, F. et al. Inflammation, fracture and bone repair. Bone 86, 119–130. https://doi.org/10.1016/j.bone.2016.02.020 (2016).

2. Redlich, K. & Smolen, J. S. Inflammatory bone loss: pathogenesis and therapeutic intervention. Nat. Rev. Drug Discovery 11, 234–250. https://doi.org/10.1038/nrd3669 (2012).

3. Mohsin, S. et al. An update on therapies for the treatment of diabetes-induced osteoporosis. Expert Opin. Biol. Ther. 19, 937–948. https://doi.org/10.1080/14712598.2019.1618266 (2019).

4. Tsukamoto, M. et al. Elcatonin prevents bone loss caused by skeletal unloading by inhibiting preosteoclast fusion through the unloading-induced high expression of calcitonin receptors in bone marrow cells. Bone 85, 70–80. https://doi.org/10.1016/j.bone.2016.01.025 (2016).

5. Chen, Z. et al. The role of histone deacetylase 4 during chondrocyte hypertrophy and endochondral bone development. Bone Jt. Res. 9, 82–89. https://doi.org/10.1302/2046-3758.92.Bjr-2019-0172.R1 (2020).

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