RNA SEQ Analysis Indicates that the AE3 Cl−/HCO3 − Exchanger Contributes to Active Transport-Mediated CO2 Disposal in Heart
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-07585-y.pdf
Reference140 articles.
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2. Sowah, D., Brown, B. F., Quon, A., Alvarez, B. V. & Casey, J. R. Resistance to cardiomyocyte hypertrophy in ae3−/− mice, deficient in the AE3 Cl−/HCO3 − exchanger. BMC Cardiovasc Disord 14, 89, doi: 10.1186/1471-2261-14-89 (2014).
3. Prasad, V. et al. Impaired cardiac contractility in mice lacking both the AE3 Cl−/HCO3 − exchanger and the NKCC1 Na+-K+-2Cl− cotransporter: effects on Ca2+ handling and protein phosphatases. J Biol Chem 283, 31303–31314, doi: 10.1074/jbc.M803706200 (2008).
4. Prasad, V. et al. Loss of the AE3 Cl−/HCO3 − exchanger in mice affects rate-dependent inotropy and stress-related AKT signaling in heart. Front Physiol 4, 399, doi: 10.3389/fphys.2013.00399 (2013).
5. Al Moamen, N. J. et al. Loss of the AE3 anion exchanger in a hypertrophic cardiomyopathy model causes rapid decompensation and heart failure. J Mol Cell Cardiol 50, 137–146, doi: 10.1016/j.yjmcc.2010.10.028 (2011).
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