Deletion of NFKB1 enhances canonical NF-κB signaling and increases macrophage and myofibroblast content during tendon healing-Reference-Cited by-同舟云学术

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Deletion of NFKB1 enhances canonical NF-κB signaling and increases macrophage and myofibroblast content during tendon healing

Published:2019-07-29 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Best Katherine T.,Lee Fredella K.,Knapp Emma,Awad Hani A.,Loiselle Alayna E.^ORCID

Funder

U.S. Department of Health & Human Services | NIH | National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-47461-5.pdf

Reference35 articles.

1. Beredjiklian, P. K. et al. Regenerative versus reparative healing in tendon: a study of biomechanical and histological properties in fetal sheep. Annals of biomedical engineering 31, 1143–1152 (2003).

2. Favata, M. et al. Regenerative properties of fetal sheep tendon are not adversely affected by transplantation into an adult environment. Journal of orthopaedic research: official publication of the Orthopaedic Research Society 24, 2124–2132, https://doi.org/10.1002/jor.20271 (2006).

3. Manning, C. N. et al. The early inflammatory response after flexor tendon healing: a gene expression and histological analysis. Journal of orthopaedic research: official publication of the Orthopaedic Research Society 32, 645–652, https://doi.org/10.1002/jor.22575 (2014).

4. Hayden, M. S. & Ghosh, S. NF-kappaB, the first quarter-century: remarkable progress and outstanding questions. Genes & development 26, 203–234, https://doi.org/10.1101/gad.183434.111 (2012).

5. Chen, S. et al. RelA/p65 inhibition prevents tendon adhesion by modulating inflammation. cell proliferation, and apoptosis. Cell death & disease 8, e2710, https://doi.org/10.1038/cddis.2017.135 (2017).

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