Functional analysis of SLC39A8 mutations and their implications for manganese deficiency and mitochondrial disorders
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-21464-0.pdf
Reference41 articles.
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2. Milatovic, D., Zaja-Milatovic, S., Gupta, R. C., Yu, Y. & Aschner, M. Oxidative damage and neurodegeneration in manganese-induced neurotoxicity. Toxicology and applied pharmacology 240, 219–225, https://doi.org/10.1016/j.taap.2009.07.004 (2009).
3. Stanwood, G. D. et al. Manganese exposure is cytotoxic and alters dopaminergic and GABAergic neurons within the basal ganglia. Journal of neurochemistry 110, 378–389, https://doi.org/10.1111/j.1471-4159.2009.06145.x (2009).
4. Jeong, J. & Eide, D. J. The SLC39 family of zinc transporters. Molecular aspects of medicine 34, 612–619, https://doi.org/10.1016/j.mam.2012.05.011 (2013).
5. He, L. et al. ZIP8, member of the solute-carrier-39 (SLC39) metal-transporter family: characterization of transporter properties. Molecular pharmacology 70, 171–180, https://doi.org/10.1124/mol.106.024521 (2006).
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