Insulin, glucagon and somatostatin stores in the pancreas of subjects with type-2 diabetes and their lean and obese non-diabetic controls
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-10296-z.pdf
Reference49 articles.
1. Ferrannini, E. et al. β-Cell function in subjects spanning the range from normal glucose tolerance to overt diabetes: a new analysis. J. Clin. Endocrinol. Metab. 90, 493–500 (2005).
2. Dunning, B. E. & Gerich, J. E. The role of α-cell dysregulation in fasting and postprandial hyperglycemia in type 2 diabetes and therapeutic implications. Endocr. Rev. 28, 253–283 (2007).
3. D’Alessio, D. The role of dysregulated glucagon secretion in type 2 diabetes. Diabetes Obes. Metab. 13(Suppl 1), 126–132 (2011).
4. Scott, D. A. & Fischer, A. M. The insulin and zinc content of normal and diabetic pancreas. J. Clin. Invest. 17, 727–731 (1938).
5. Wrenshall, G. A., Bogoch, A. & Ritchie, R. C. Extractable insulin of pancreas; correlation with pathological and clinical findings in diabetic and nondiabetic cases. Diabetes 1, 87–107 (1952).
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