IL-33-induced reactive oxygen species are required for optimal metabolic programming in group 2 innate lymphoid cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Immunology,Immunology and Allergy
Link
http://www.nature.com/articles/s41423-020-0393-z.pdf
Reference8 articles.
1. Henricks, P. A. & Nijkamp, F. P. Reactive oxygen species as mediators in asthma. Pulm. Pharmacol. Ther. 14, 409–420 (2001).
2. Dozor, A. J. The role of oxidative stress in the pathogenesis and treatment of asthma. Ann. N. Y. Acad. Sci. 1203, 133–137 (2010).
3. Salmond, R. J. et al. IL-33 induces innate lymphoid cell-mediated airway inflammation by activating mammalian target of rapamycin. J. Allergy Clin. Immunol. 130, 1159–1166 (2012).
4. Lam, G. Y., Huang, J. & Brumell, J. H. The many roles of NOX2 NADPH oxidase-derived ROS in immunity. Semin. Immunopathol. 32, 415–430 (2010).
5. Vlahos, R. et al. Inhibition of Nox2 oxidase activity ameliorates influenza A virus-induced lung inflammation. PLoS Pathog. 7, e1001271 (2011).
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