Differential cell proliferation and cell death during the urethral groove formation in guinea pig model
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology and Child Health
Link
http://www.nature.com/articles/s41390-018-0236-0.pdf
Reference28 articles.
1. Nelson, C. P. et al. The increasing incidence of congenital penile anomalies in the United States. J. Urol. 174, 1573–1576 (2005).
2. Bouty, A., Ayers, K. L., Pask, A., Heloury, Y. & Sinclair, A. H. The genetic and environmental factors underlying hypospadias. Sex. Dev. 9, 239–259 (2015).
3. Paulozzi, L. J., Erickson, J. D. & Jackson, R. J. Hypospadias trends in two US surveillance systems. Pediatrics 100, 831–834 (1997).
4. Nordenvall, A. S., Frisen, L., Nordenstrom, A., Lichtenstein, P. & Nordenskjold, A. Population based nationwide study of hypospadias in Sweden, 1973 to 2009: incidence and risk factors. J. Urol. 191, 783–789 (2014).
5. Kalfa, N., Philibert, P., Baskin, L. S. & Sultan, C. Hypospadias: interactions between environment and genetics. Mol. Cell. Endocrinol. 335, 89–95 (2011).
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2. Etiology of Hypospadias: A Comparative Review of Genetic Factors and Developmental Processes Between Human and Animal Models;Research and Reports in Urology;2020-12
3. Insights Figure for “Differential cell proliferation and cell death during the urethral groove formation in guinea pig model”;Pediatric Research;2019-06-18
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