Dopamine D1R-neuron cacna1c deficiency: a new model of extinction therapy-resistant post-traumatic stress-Reference-Cited by-同舟云学术

Dopamine D1R-neuron cacna1c deficiency: a new model of extinction therapy-resistant post-traumatic stress

Published:2020-04-24 Issue:6 Volume:26 Page:2286-2298
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Bavley Charlotte C.,Kabir Zeeba D.,Walsh Alexander P.,Kosovsky Maria,Hackett Jonathan,Sun Herie,Vázquez-Rosa Edwin,Cintrón-Pérez Coral J.,Miller Emiko,Koh Yeojung,Pieper Andrew A.,Rajadhyaksha Anjali M.

Funder

U.S. Department of Health & Human Services | NIH | National Institute on Drug Abuse

Hartwell Foundation

U.S. Department of Health & Human Services | NIH | National Center for Advancing Translational Sciences

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

https://www.nature.com/articles/s41380-020-0730-8.pdf

Reference58 articles.

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4. Logue MW, van Rooij SJH, Dennis EL, Davis SL, Hayes JP, Stevens JS, et al. Smaller hippocampal volume in posttraumatic stress disorder: a multisite ENIGMA-PGC study: subcortical volumetry results from posttraumatic stress disorder consortia. Biol Psychiatry. 2018;83:244–53.

5. van Rooij SJH, Stevens JS, Ely TD, Hinrichs R, Michopoulos V, Winters SJ, et al. The role of the hippocampus in predicting future posttraumatic stress disorder symptoms in recently traumatized civilians. Biol Psychiatry. 2017;84:106–15.

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