How autism and Alzheimer’s disease are TrAPPed
Author:
Funder
U.S. Department of Health & Human Services | NIH | National Institute on Aging
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology
Link
http://www.nature.com/articles/s41380-020-00928-8.pdf
Reference21 articles.
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2. Mosconi MW, Cody-Hazlett H, Poe MD, Gerig G, Gimpel-Smith R, Piven J. Longitudinal study of amygdala volume and joint attention in 2- to 4-year-old children with autism. Arch Gen Psychiatry. 2009;66:509–16.
3. Mattson MP. Secreted forms of beta-amyloid precursor protein modulate dendrite outgrowth and calcium responses to glutamate in cultured embryonic hippocampal neurons. J Neurobiol. 1994;25:439–50.
4. Tsatsanis A, Wong BX, Gunn AP, Ayton S, Bush AI, Devos D, et al. Amyloidogenic processing of Alzheimer’s disease beta-amyloid precursor protein induces cellular iron retention. Mol Psychiatry. 2020;25:1958–66.
5. Ray B, Long JM, Sokol DK, Lahiri DK. Increased secreted amyloid precursor protein-alpha (sAPPalpha) in severe autism: proposal of a specific, anabolic pathway and putative biomarker. PLoS One. 2011;6:e20405.
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