Blunted apoptosis of erythrocytes in mice deficient in the heterotrimeric G-protein subunit Gαi2-Reference-Cited by-同舟云学术

Blunted apoptosis of erythrocytes in mice deficient in the heterotrimeric G-protein subunit Gαi2

Published:2016-08 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Bissinger Rosi,Lang Elisabeth,Ghashghaeinia Mehrdad,Singh Yogesh,Zelenak Christine,Fehrenbacher Birgit,Honisch Sabina,Chen Hong,Fakhri Hajar,Umbach Anja T.,Liu Guilai,Rexhepaj Rexhep,Liu Guoxing,Schaller Martin,Mack Andreas F.,Lupescu Adrian,Birnbaumer Lutz,Lang Florian,Qadri Syed M.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep30925.pdf

Reference60 articles.

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2. Wettschureck, N. & Offermanns, S. Mammalian G proteins and their cell type specific functions. Physiol Rev 85, 1159–1204 (2005).

3. Simon, M. I., Strathmann, M. P. & Gautam, N. Diversity of G proteins in signal transduction. Science 252, 802–808 (1991).

4. Devanathan, V. et al. Platelet Gi protein Galphai2 is an essential mediator of thrombo-inflammatory organ damage in mice. Proc. Natl. Acad. Sci. USA 112, 6491–6496 (2015).

5. Pero, R. S. et al. Galphai2-mediated signaling events in the endothelium are involved in controlling leukocyte extravasation. Proc. Natl. Acad. Sci. USA 104, 4371–4376 (2007).

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