Comparative metabolomics profiling of isogenic KRAS wild type and mutant NSCLC cells in vitro and in vivo
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep28398.pdf
Reference14 articles.
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2. Vizan, P., Boros, L. G., Figueras, A., Capella, G., Mangues, R. et al. K-ras codon-specific mutations produce distinctive metabolic phenotypes in NIH3T3 mice [corrected] fibroblasts. Cancer Res 65, 5512–5 (2005).
3. Gaglio, D., Metallo, C. M., Gameiro, P. A., Hiller, K., Danna, L. S. et al. Oncogenic K-Ras decouples glucose and glutamine metabolism to support cancer cell growth. Mol Syst Biol 7, 523 (2011).
4. Son, J., Lyssiotis, C. A., Ying, H., Wang, X., Hua, S. et al. Glutamine supports pancreatic cancer growth through a KRAS-regulated metabolic pathway. Nature 496, 101–5 (2013).
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