Agonist Activated PKCβII Translocation and Modulation of Cardiac Myocyte Contractile Function-Reference-Cited by-同舟云学术

Agonist Activated PKCβII Translocation and Modulation of Cardiac Myocyte Contractile Function

Published:2013-06-12 Issue:1 Volume:3 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Hwang Hyosook,Robinson Dustin,Rogers Julie B.,Stevenson Tamara K.,Lang Sarah E.,Sadayappan Sakthivel,Day Sharlene M.,Sivaramakrishnan Sivaraj,Westfall Margaret V.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep01971.pdf

Reference43 articles.

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3. Takeishi, Y., Chu, G. X., Kirkpatrick, D. M., Li, Z. L., Wakasaki, H., Kranias, E. G., King, G. L. & Walsh, R. A. In vivo phosphorylation of cardiac troponin I by protein kinase C β2 decreases cardiomyocyte calcium responsiveness and contractility in transgenic mouse hearts. J Clin Invest 102, 72–78 (1998).

4. Gu, X. & Bishop, S. P. Increased Protein-Kinase-C and Isozyme Redistribution in Pressure-Overload Cardiac-Hypertrophy in the Rat. Circ Res 75, 926–931 (1994).

5. Hambleton, M., Hahn, H., Pleger, S. T., Kuhn, M. C., Klevitsky, R., Carr, A. N., Kimball, T. F., Hewett, T. E., Dorn, G. W., 2nd, Koch, W. J. & Molkentin, J. D. Pharmacological- and gene therapy-based inhibition of protein kinase Calpha/beta enhances cardiac contractility and attenuates heart failure. Circulation 114, 574–582 (2006).

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