53BP1 promotes non-homologous end joining of telomeres by increasing chromatin mobility
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/nature07433.pdf
Reference32 articles.
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2. Celli, G. B., Lazzerini Denchi, E. & de Lange, T. Ku70 stimulates fusion of dysfunctional telomeres yet protects chromosome ends from homologous recombination. Nature Cell Biol. 8, 855–890 (2006)
3. Dimitrova, N. & de Lange, T. MDC1 accelerates non-homologous end-joining of dysfunctional telomeres. Genes Dev. 20, 3238–3243 (2006)
4. Lazzerini Denchi, E. & de Lange, T. Protection of telomeres through independent control of ATM and ATR by TRF2 and POT1. Nature 448, 1068–1071 (2007)
5. DiTullio, R. A. et al. 53BP1 functions in an ATM-dependent checkpoint pathway that is constitutively activated in human cancer. Nature Cell Biol. 4, 998–1002 (2002)
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