Axonal transport deficits and neurodegenerative diseases
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://www.nature.com/articles/nrn3380.pdf
Reference179 articles.
1. Perlson, E., Maday, S., Fu, M. M., Moughamian, A. J. & Holzbaur, E. L. Retrograde axonal transport: pathways to cell death? Trends Neurosci. 33, 335–344 (2010).
2. Roy, S. et al. Neurofilaments are transported rapidly but intermittently in axons: implications for slow axonal transport. J. Neurosci. 20, 6849–6861 (2000).
3. Wang, L., Ho, C. L., Sun, D., Liem, R. K. & Brown, A. Rapid movement of axonal neurofilaments interrupted by prolonged pauses. Nature Cell Biol. 2, 137–141 (2000). References 2 and 3 show that the slow axonal transport rate of neurofilaments is the result of rapid movements and prolonged pauses.
4. Maday, S., Wallace, K. E. & Holzbaur, E. L. Autophagosomes initiate distally and mature during transport toward the cell soma in primary neurons. J. Cell Biol. 196, 407–417 (2012).
5. Millecamps, S., Gowing, G., Corti, O., Mallet, J. & Julien, J. P. Conditional NF-L transgene expression in mice for in vivo analysis of turnover and transport rate of neurofilaments. J. Neurosci. 27, 4947–4956 (2007). This study shows that the axonal transport rate of cytoskeleton components in vivo depends on the density of the stationary network in the axons.
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