Nuclear pore complexes — a doorway to neural injury in neurodegeneration
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical)
Link
https://www.nature.com/articles/s41582-022-00653-6.pdf
Reference188 articles.
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2. Raices, M. & D’Angelo, M. A. Nuclear pore complex composition: a new regulator of tissue-specific and developmental functions. Nat. Rev. Mol. Cell Biol. 13, 687–699 (2012).
3. Raices, M. & D’Angelo, M. A. Nuclear pore complexes and regulation of gene expression. Curr. Opin. Cell Biol. 46, 26–32 (2017).
4. Chou, C. C. et al. TDP43 pathology disrupts nuclear pore complexes and nucleocytoplasmic transport in ALS/FTD. Nat. Neurosci. 21, 228–239 (2018).
5. Coyne, A. N. et al. G(4)C(2) repeat RNA initiates a POM121-mediated reduction in specific nucleoporins in C9orf72 ALS/FTD. Neuron https://doi.org/10.1016/j.neuron.2020.06.027 (2020). The first study to document a specific NPC defect in neurodegeneration and how this nucleoporin defect can initiate NPC injury.
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