MiR-361-3p regulates ERK1/2-induced EMT via DUSP2 mRNA degradation in pancreatic ductal adenocarcinoma-Reference-Cited by-同舟云学术

MiR-361-3p regulates ERK1/2-induced EMT via DUSP2 mRNA degradation in pancreatic ductal adenocarcinoma

Published:2018-07-24 Issue:8 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Hu Jisheng,Li Le,Chen Hongze,Zhang Guangquan,Liu Huan,Kong Rui,Chen Hua,Wang Yongwei,Li Yilong,Tian Fengyu,Lv Xinjian,Li Guanqun,Sun Bei

Funder

National Natural Science Foundation of China (National Science Foundation of China)

China Postdoctoral Science Foundation

The Heilongjiang Providence Postdoctoral Science Foundation (N/A)

Heilongjiang Postdoctoral Scientific Research Developmental Fund (LBH-Q16180) The Science and Technology Project of Heilongjiang Educational Committee (12511246)

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0839-8.pdf

Reference51 articles.

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3. Rhim, A. D. et al. EMT and dissemination precede pancreatic tumor formation. Cell 148, 349–361 (2012).

4. Zheng, X. et al. Epithelial-to-mesenchymal transition is dispensable for metastasis but induces chemoresistance in pancreatic cancer. Nature 527, 525–530 (2015).

5. Aiello, N. M. et al. Upholding a role for EMT in pancreatic cancer metastasis. Nature 547, E7–E8 (2017).

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