DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF‐κB-Reference-Cited by-同舟云学术

DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF‐κB

Published:2009-07-10 Issue:8 Volume:10 Page:916-922
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Rebsamen Manuele¹,Heinz Leonhard X¹,Meylan Etienne¹,Michallet Marie‐Cécile¹,Schroder Kate¹,Hofmann Kay²,Vazquez Jessica¹,Benedict Chris A³,Tschopp Jürg¹

Affiliation:

1. Department of Biochemistry, University of Lausanne CH‐1066 Epalinges Switzerland

2. Miltenyi Biotec GmbH D‐50829 Koeln Germany

3. Division of Molecular Immunology, La Jolla Institute for Allergy & Immunology La Jolla California 92037 USA

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1038/embor.2009.109

Reference25 articles.

1. A Ribonucleotide Reductase Homolog of Cytomegalovirus and Endothelial Cell Tropism

2. An orthogonal proteomic-genomic screen identifies AIM2 as a cytoplasmic DNA sensor for the inflammasome

3. AIM2 activates the inflammasome and cell death in response to cytoplasmic DNA

4. AIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASC

5. A Toll-like receptor–independent antiviral response induced by double-stranded B-form DNA

Cited by 292 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Low levels of supercoiled mitochondrial DNA are involved in heart failure induced by transverse aortic constriction in mice via an inflammatory response mediated by ZBP1;Experimental Cell Research;2024-09

2. Necroptosis and autoimmunity;Clinical Immunology;2024-09

3. Z-DNA formation in promoters conserved between human and mouse are associated with increased transcription reinitiation rates;Scientific Reports;2024-08-01

4. Host Innate Antiviral Response to Influenza A Virus Infection: From Viral Sensing to Antagonism and Escape;Pathogens;2024-07-03

5. ZBP1 causes inflammation by inducing RIPK3-mediated necroptosis and RIPK1 kinase activity-independent apoptosis;Cell Death & Differentiation;2024-06-07