Differential activity of GSK-3 isoforms regulates NF-κB and TRAIL- or TNFα induced apoptosis in pancreatic cancer cells-Reference-Cited by-同舟云学术

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Differential activity of GSK-3 isoforms regulates NF-κB and TRAIL- or TNFα induced apoptosis in pancreatic cancer cells

Published:2014-03 Issue:3 Volume:5 Page:e1142-e1142
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Zhang J-S,Herreros-Villanueva M,Koenig A,Deng Z,de Narvajas A A-M,Gomez T S,Meng X,Bujanda L,Ellenrieder V,Li X K,Kaufmann S H,Billadeau D D

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2014102.pdf

Reference57 articles.

1. Fulda S . Apoptosis pathways and their therapeutic exploitation in pancreatic cancer. J Cell Mol Med 2009; 13: 1221–1227.

2. Fulda S, Debatin KM . Exploiting death receptor signaling pathways for tumor therapy. Biochim Biophys Acta 2004; 1705: 27–41.

3. Micheau O, Shirley S, Dufour F . Death receptors as targets in cancer. Br J Pharmacol 2013; 169: 1723–1744.

4. Ashkenazi A . Targeting the extrinsic apoptosis pathway in cancer. Cytokine Growth Factor Rev 2008; 19: 325–331.

5. Kruyt FA . TRAIL and cancer therapy. Cancer Lett 2008; 263: 14–25.

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