Control of adipose tissue cellularity by the terminal complement cascade
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://www.nature.com/articles/s41574-023-00900-w.pdf
Reference10 articles.
1. Kruglikov, I. L., Shah, M. & Scherer, P. E. Obesity and diabetes as comorbidities for COVID-19: underlying mechanisms and the role of viral-bacterial interactions. eLife 9, e61330 (2020).
2. Mathieson, P. W., Würzner, R., Oliveria, D. B., Lachmann, P. J. & Peters, D. K. Complement-mediated adipocyte lysis by nephritic factor sera. J. Exp. Med. 177, 1827–1831 (1993).
3. van Greevenbroek, M. M. J. et al. Up-regulation of the complement system in subcutaneous adipocytes from nonobese, hypertriglyceridemic subjects is associated with adipocyte insulin resistance. J. Clin. Endocrinol. Metab. 97, 4742–4752 (2012).
4. Kaye, S. et al. Upregulation of early and downregulation of terminal pathway complement genes in subcutaneous adipose tissue and adipocytes in acquired obesity. Front. Immunol. 8, 545 (2017).
5. Corvillo, F. et al. Complement factor D (adipsin) levels are elevated in acquired partial lipodystrophy (Barraquer–Simons syndrome). Int. J. Mol. Sci. 22, 6608 (2021).
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