Calorie restriction and rapamycin distinctly mitigate aging-associated protein phosphorylation changes in mouse muscles-Reference-Cited by-同舟云学术

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Calorie restriction and rapamycin distinctly mitigate aging-associated protein phosphorylation changes in mouse muscles

Published:2024-08-10 Issue:1 Volume:7 Page:
ISSN:2399-3642
Container-title:Communications Biology
language:en
Short-container-title:Commun Biol

Author:

Ataman Meric^ORCID,Mittal Nitish^ORCID,Tintignac Lionel^ORCID,Schmidt Alexander^ORCID,Ham Daniel J.^ORCID,González Asier^ORCID,Ruegg Markus A.^ORCID,Zavolan Mihaela^ORCID

Funder

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

Jubiläumsstiftung von Swiss Life

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s42003-024-06679-4.pdf

Reference75 articles.

1. Niccoli, T. & Partridge, L. Ageing as a risk factor for disease. Curr. Biol. 22, R741–R752 (2012).

2. Siparsky, P. N., Kirkendall, D. T. & Garrett, W. E. Jr. Muscle changes in aging: understanding sarcopenia. Sports Health 6, 36–40 (2014).

3. Evans, W. J. Skeletal muscle loss: cachexia, sarcopenia, and inactivity. Am. J. Clin. Nutr. 91, 1123S–1127S (2010).

4. Sandri, M. et al. Signalling pathways regulating muscle mass in ageing skeletal muscle: the role of the IGF1-Akt-mTOR-FoxO pathway. Biogerontology 14, 303–323 (2013).

5. Ham, D. J. et al. The neuromuscular junction is a focal point of mTORC1 signaling in sarcopenia. Nat. Commun. 11, 4510 (2020).

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