Coexistence of different phenotypes in a family with glucocorticoid-remediable aldosteronism-Reference-Cited by-同舟云学术

Coexistence of different phenotypes in a family with glucocorticoid-remediable aldosteronism

Published:2003-12-17 Issue:1 Volume:18 Page:47-51
ISSN:0950-9240
Container-title:Journal of Human Hypertension
language:en
Short-container-title:J Hum Hypertens

Author:

Fallo F,Pilon C,Williams T A,Sonino N,Morra di Cella S,Veglio F,De Iasio R,Montanari P,Mulatero P

Publisher

Springer Science and Business Media LLC

Subject

Internal Medicine

Link

http://www.nature.com/articles/1001636.pdf

Reference22 articles.

1. Sutherland DJA, Ruse JL, Laidlaw JC . Hypertension, increased aldosterone secretion and low plasma renin activity relieved by dexamethasone. Can Med Assoc J 1966; 95: 1109–1119.

2. Dluhy RG, Lifton RF . Glucocorticoid-remediable aldosteronism. J Clin Endocrinol Metab 1999; 84: 4341–4344.

3. Stowasser M, Gordon RD . Primary aldosteronism: learning from the study of familial varieties. J Hypertens 2000; 18: 1165–1176.

4. Lifton RB et al. A chimaeric 11β-hydroxylase/aldosterone synthase gene causes glucocorticoid remediable aldosteronism and human hypertension. Nature 1992; 35: 262–265.

5. Mulatero P et al. Diagnosis of glucocorticoid-remediable aldosteronism in primary aldosteronism: aldosterone response to dexamethasone and long polymerase chain reaction for chimeric gene. J Clin Endocrinol Metab 1998; 83: 2573–2575.

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