CD69 controls the uptake of L-tryptophan through LAT1-CD98 and AhR-dependent secretion of IL-22 in psoriasis-Reference-Cited by-同舟云学术

CD69 controls the uptake of L-tryptophan through LAT1-CD98 and AhR-dependent secretion of IL-22 in psoriasis

Published:2016-07-04 Issue:8 Volume:17 Page:985-996
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Cibrian Danay,Saiz María Laura,de la Fuente Hortensia,Sánchez-Díaz Raquel,Moreno-Gonzalo Olga,Jorge Inmaculada,Ferrarini Alessia,Vázquez Jesús,Punzón Carmen,Fresno Manuel,Vicente-Manzanares Miguel,Daudén Esteban,Fernández-Salguero Pedro M,Martín Pilar^ORCID,Sánchez-Madrid Francisco

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/ni.3504.pdf

Reference49 articles.

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2. Lowes, M.A., Suárez-Fariñas, M. & Krueger, J.G. Immunology of psoriasis. Annu. Rev. Immunol. 32, 227–255 (2014).

3. Leonardi, C. et al. Anti-interleukin-17 monoclonal antibody ixekizumab in chronic plaque psoriasis. N. Engl. J. Med. 366, 1190–1199 (2012).

4. Chan, J.R. et al. IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2-dependent mechanisms with implications for psoriasis pathogenesis. J. Exp. Med. 203, 2577–2587 (2006).

5. Zheng, Y. et al. Interleukin-22, a TH17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis. Nature 445, 648–651 (2007).

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