CBFβ-SMMHC regulates ribosomal gene transcription and alters ribosome biogenesis-Reference-Cited by-同舟云学术

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CBFβ-SMMHC regulates ribosomal gene transcription and alters ribosome biogenesis

Published:2017-02-15 Issue:6 Volume:31 Page:1443-1446
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Cordonnier G,Mandoli A,Radhouane A,Hypolite G,Lhermitte L,Belhocine M,Asnafi V,Macintyre E,Martens J H A,Fumagalli S,Bond J

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

https://www.nature.com/articles/leu201753.pdf

Reference15 articles.

1. Young DW, Hassan MQ, Pratap J, Galindo M, Zaidi SK, Lee SH et al. Mitotic occupancy and lineage-specific transcriptional control of rRNA genes by Runx2. Nature 2007; 445: 442–446.

2. Ali SA, Zaidi SK, Dacwag CS, Salma N, Young DW, Shakoori AR et al. Phenotypic transcription factors epigenetically mediate cell growth control. Proc Natl Acad Sci USA 2008; 105: 6632–6637.

3. Cai X, Gao L, Teng L, Ge J, Oo ZM, Kumar AR et al. Runx1 deficiency decreases ribosome biogenesis and confers stress resistance to hematopoietic stem and progenitor cells. Cell Stem Cell 2015; 17: 165–177.

4. Bakshi R, Zaidi SK, Pande S, Hassan MQ, Young DW, Montecino M et al. The leukemogenic t(8;21) fusion protein AML1-ETO controls rRNA genes and associates with nucleolar-organizing regions at mitotic chromosomes. J Cell Sci 2008; 121 (Pt 23): 3981–3990.

5. Lopez-Camacho C, van Wijnen AJ, Lian JB, Stein JL, Stein GS . CBFbeta and the leukemogenic fusion protein CBFbeta-SMMHC associate with mitotic chromosomes to epigenetically regulate ribosomal genes. J Cell Biochem 2014; 115: 2155–2164.

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2. Emerging therapies for inv(16) AML;Blood;2021-05-13

3. The RUNX1/RUNX1T1 network: translating insights into therapeutic options;Experimental Hematology;2021-02

4. CBFβ-SMMHC Affects Genome-wide Polycomb Repressive Complex 1 Activity in Acute Myeloid Leukemia;Cell Reports;2020-01

5. CBFβ-MYH11 interferes with megakaryocyte differentiation via modulating a gene program that includes GATA2 and KLF1;Blood Cancer Journal;2019-03

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