Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin-Reference-Cited by-同舟云学术

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Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin

Published:2006-05-03 Issue:7097 Volume:441 Page:1157-1161
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Park Jeehye,Lee Sung Bae,Lee Sungkyu,Kim Yongsung,Song Saera,Kim Sunhong,Bae Eunkyung,Kim Jaeseob,Shong Minho,Kim Jin-Man,Chung Jongkyeong

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature04788.pdf

Reference18 articles.

1. Shen, J. & Cookson, M. R. Mitochondria and dopamine: new insights into recessive parkinsonism. Neuron 43, 301–304 (2004)

2. Moore, D. J., West, A. B., Dawson, V. L. & Dawson, T. M. Molecular pathophysiology of Parkinson's disease. Annu. Rev. Neurosci. 28, 57–87 (2005)

3. Unoki, M. & Nakamura, Y. Growth-suppressive effects of BPOZ and EGR2, two genes involved in the PTEN signaling pathway. Oncogene 20, 4457–4465 (2001)

4. Valente, E. M. et al. Hereditary early-onset Parkinson's disease caused by mutations in PINK1. Science 304, 1158–1160 (2004)

5. Vander Heiden, M. G., Chandel, N. S., Williamson, E. K., Schumacker, P. T. & Thompson, C. B. Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria. Cell 91, 627–637 (1997)

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