Complement C3 activation regulates the production of tRNA-derived fragments Gly-tRFs and promotes alcohol-induced liver injury and steatosis-Reference-Cited by-同舟云学术

Complement C3 activation regulates the production of tRNA-derived fragments Gly-tRFs and promotes alcohol-induced liver injury and steatosis

Published:2019-05-10 Issue:7 Volume:29 Page:548-561
ISSN:1001-0602
Container-title:Cell Research
language:en
Short-container-title:Cell Res

Author:

Zhong Fudi,Hu Zhigao,Jiang Keqing,Lei Biao,Wu Zhan,Yuan Guandou,Luo Hongliang,Dong Chunqiang,Tang Bo,Zheng Chaowen,Yang Shuai,Zeng Yonglian,Guo Zhenya,Yu Shuiping,Su Huizhao,Zhang Guo,Qiu Xiaoqiang,Tomlinson Stephen,He Songqing

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/s41422-019-0175-2.pdf

Reference69 articles.

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3. Rehm, J. et al. Global burden of disease and injury and economic cost attributable to alcohol use and alcohol-use disorders. Lancet 373, 2223–33 (2009).

4. Pritchard, M. T. et al. Differential contributions of C3, C5, and decay-accelerating factor to ethanol-induced fatty liver in mice. Gastroenterology 132, 1117–26 (2007).

5. Roychowdhury, S. et al. An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice. Hepatology 49, 1326–34 (2009).

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