Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formation-Reference-Cited by-同舟云学术

Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formation

Published:1999-05 Issue:5 Volume:5 Page:560-564
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Chui De-Hua,Tanahashi Hiroshi,Ozawa Kazuharu,Ikeda Sachiya,Checler Frédéric,Ueda Otoya,Suzuki Hiroshi,Araki Wataru,Inoue Haruhisa,Shirotani Keiro,Takahashi Keikichi,Gallyas Ferenc,Tabira Takeshi

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm0599_560.pdf

Reference29 articles.

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2. Cruts, M., Van Broeckhoven, C. Presenilin mutations in Alzheimer's disease. Hum. Mutat. 11, 183 –190 (1998).

3. Kamimura, K. et al. Familial Alzheimer's disease genes in Japanese. J. Neurol. Sci. 160, 76–81 (1998).

4. Scheuner, D. et al. The amyloid β-protein deposited in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease. Nature Med. 2, 864–870 (1996).

5. Lemere, C. A. et al. The E280A presenilin-1 Alzheimer mutation produces increased Aβ42 deposition and severe cerebellar pathology. Nature Med. 2, 1146–1150 (1996).

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