FRA3B and other common fragile sites: the weakest links
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Earth and Planetary Sciences,General Environmental Science
Link
http://www.nature.com/articles/35106058.pdf
Reference82 articles.
1. Cohen, A. J. et al. Hereditary renal-cell carcinoma associated with a chromosomal translocation. N. Engl. J. Med. 301, 592–595 (1979).
2. Glover, T. W., Berger, C., Coyle, J. & Echo, B. DNA polymerase-α inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes. Hum. Genet. 67, 136–142 (1984).Reports that the breaks and gaps induced by aphidicolin represent a new class of fragile sites: the common fragile sites.
3. Yunis, J. J. & Soreng, A. L. Constitutive fragile sites and cancer. Science 226, 1199–1204 (1984).Notes that locations of about half of the common fragile sites seem cytogenetically to coincide with locations of specific chromosome translocations that are associated with human cancers and might be near proto-oncogenes.
4. Ohta, M. et al. The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers. Cell 84, 587–597 (1996).Reports the cloning of the FHIT gene, the t(3;8) translocation, the FRA3B fragile site and homozygous deletions in cancer cells.
5. Zimonjic, D. B. et al. Positions of chromosome 3p14.2 fragile sites (FRA3B) within the FHIT gene. Cancer Res. 57, 1166–1170 (1997).
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