Effect of the BTK inhibitor ibrutinib on macrophage- and γδ T cell-mediated response against Mycobacterium tuberculosis-Reference-Cited by-同舟云学术

Effect of the BTK inhibitor ibrutinib on macrophage- and γδ T cell-mediated response against Mycobacterium tuberculosis

Published:2018-11 Issue:11 Volume:8 Page:
ISSN:2044-5385
Container-title:Blood Cancer Journal
language:en
Short-container-title:Blood Cancer Journal

Author:

Colado Ana,Genoula Melanie,Cougoule Céline,Marín Franco José L.,Almejún María B.,Risnik Denise,Kviatcovsky Denise,Podaza Enrique,Elías Esteban E.,Fuentes Federico,Maridonneau-Parini Isabelle,Bezares Fernando R.,Fernandez Grecco Horacio,Cabrejo María,Jancic Carolina,Sasiain María del Carmen,Giordano Mirta,Gamberale Romina,Balboa Luciana,Borge Mercedes^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Hematology

Link

http://www.nature.com/articles/s41408-018-0136-x.pdf

Reference15 articles.

1. Dubovsky, J. A. et al. Ibrutinib is an irreversible molecular inhibitor of ITK driving a Th1-selective pressure in T lymphocytes. Blood 122, 2539–2549 (2013).

2. Fiorcari, S. et al. Ibrutinib modifies the function of monocyte/macrophage population in chronic lymphocytic leukemia. Oncotarget 7, 65968–65981 (2016).

3. Da Roit, F. et al. Ibrutinib interferes with the cell-mediated anti-tumor activities of therapeutic CD20 antibodies: implications for combination therapy. Haematologica 100, 77–86 (2015).

4. Chen, Z. W. Immune regulation of gammadelta T cell responses in mycobacterial infections. Clin. Immunol. 116, 202–207 (2005).

5. Verreck, F. A. et al. Human IL-23-producing type 1 macrophages promote but IL-10-producing type 2 macrophages subvert immunity to (myco)bacteria. Proc. Natl Acad. Sci. USA 101, 4560–4565 (2004).

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