Host–microbe interactions rewire metabolism in a C. elegans model of leucine breakdown deficiency
Author:
Funder
U.S. Department of Health & Human Services | National Institutes of Health
Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s42255-024-01098-5.pdf
Reference71 articles.
1. Duan, Y. et al. The role of leucine and its metabolites in protein and energy metabolism. Amino Acids 48, 41–51 (2016).
2. Adeva-Andany, M. M., Lopez-Maside, L., Donapetry-Garcia, C., Fernandez-Fernandez, C. & Sixto-Leal, C. Enzymes involved in branched-chain amino acid metabolism in humans. Amino Acids 49, 1005–1028 (2017).
3. Rodriguez-Aguilera, J. C., Asencio, C., Ruiz-Ferrer, M., Vela, J. & Navas, P. Caenorhabditis elegans ubiquinone biosynthesis genes. Biofactors 18, 237–244 (2003).
4. Rauthan, M. & Pilon, M. The mevalonate pathway in C. elegans. Lipids Health Dis. 10, 243 (2011).
5. Arnold, G. L. et al. A Delphi-based consensus clinical practice protocol for the diagnosis and management of 3-methylcrotonyl CoA carboxylase deficiency. Mol. Genet. Metab. 93, 363–370 (2008).
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1. Publisher Correction: Host–microbe interactions rewire metabolism in a C. elegans model of leucine breakdown deficiency;Nature Metabolism;2024-08-15
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