Loss of Normal Profilaggrin and Filaggrin in Flaky Tail (ft/ft) Mice: an Animal Model for the Filaggrin-Deficient Skin Disease Ichthyosis Vulgaris-Reference-Cited by-同舟云学术

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Loss of Normal Profilaggrin and Filaggrin in Flaky Tail (ft/ft) Mice: an Animal Model for the Filaggrin-Deficient Skin Disease Ichthyosis Vulgaris

Published:2000-12 Issue:6 Volume:115 Page:1072-1081
ISSN:0022-202X
Container-title:Journal of Investigative Dermatology
language:en
Short-container-title:Journal of Investigative Dermatology

Author:

Presland Richard B.,Boggess Dawnalyn,Patrick Lewis S.,Hull Christopher,Fleckman Philip,Sundberg John P.

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference67 articles.

1. Ultrastructural distinction of autosomal dominant ichthyosis vulgaris and X-linked recessive ichthyosis;Anton-Lamprecht;Humangenetik,1972

2. Ultrastructural evaluation of mouse mutations;Bechtold,1999

3. Mutations in ABCC6 cause pseudoxanthoma elasticum;Bergen;Nat Genet,2000

4. Loricrin expression is coordinated with other epidermal proteins and the appearance of lipid lamellar granules in development;Bickenbach;J Invest Dermatol,1995

5. Glucosylceramide synthase activity in murine epidermis: quantitation, localization, regulation, and requirement for barrier homeostasis;Chujor;J Lipid Res,1998

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