Long-term follow-up of granulocyte colony-stimulating factor receptor mutations in patients with severe congenital neutropenia: implications for leukaemogenesis and therapy
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2141.2003.04160.x/fullpdf
Reference23 articles.
1. In vivo kinetics of murine hemopoietic stem cells;Abkowitz;Blood,2000
2. Mutations in the ELA2 gene encoding neutrophil elastase are present in most patients with sporadic severe congenital neutropenia but only in some patients with the familial form of the disease;Ancliff;Blood,2001
3. Paternal mosaicism proves the pathogenic nature of mutations in neutrophil elastase in severe congenital neutropenia;Ancliff;Blood,2002
4. Leukemic transformation in patients with severe congenital neutropenia;Banerjee;Journal of Pediatric Hematology and Oncology,2001
5. Mutations of the granulocyte-colony stimulating factor receptor in patients with severe congenital neutropenia are not required for transformation to acute myeloid leukaemia and may be a bystander phenomenon;Bernard;British Journal of Haematology,1998
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