Tissue distribution of products of the mouse decay-accelerating factor (DAF) genes. Exploitation of a Daf1 knock-out mouse and site-specific monoclonal antibodies-Reference-Cited by-同舟云学术

Tissue distribution of products of the mouse decay-accelerating factor (DAF) genes. Exploitation of a Daf1 knock-out mouse and site-specific monoclonal antibodies

Published:2001-10 Issue:2 Volume:104 Page:215-225
ISSN:0019-2805
Container-title:Immunology
language:en
Short-container-title:Immunology

Author:

Lin Feng,Fukuoka Yoshihiro,Spicer Andrew,Ohta Rieko,Okada Noriko,Harris Claire L.,Emancipator Steven N.,Medof M. Edward

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2567.2001.01287.x/fullpdf

Reference55 articles.

1. Inhibition of complement activation on the surface of cells after incorporation of decay-accelerating factor (DAF) into their membranes.

2. Deficiency of an erythrocyte membrane protein with complement regulatory activity in paroxysmal nocturnal hemoglobinuria.

3. The mechanism of action of decay-accelerating factor (DAF). DAF inhibits the assembly of C3 convertases by dissociating C2a and Bb.

4. The gene encoding decay-accelerating factor (DAF) is located in the complement-regulatory locus on the long arm of chromosome 1.

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