Chemotaxis of macrophages is abolished in the Wiskott‐Aldrich syndrome
Author:
Affiliation:
1. MRC Muscle and Cell Motility Unit,
2. Muscle and Motility Research Centre, The Randall Institute, Kings College London,
3. Molecular Haematology Unit,
4. Molecular Immunology Unit, Institute of Child Health, University College London
Publisher
Wiley
Subject
Hematology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1046/j.1365-2141.1998.00767.x
Reference43 articles.
1. Rho, Rac and Cdc42 regulate actin organization and cell adhesion in macrophages;Allen W.E.;Journal of Cell Science,1997
2. Abnormalities of chemotactic lymphokine synthesis and mononuclear leukocyte chemotaxis in Wiskott‐Aldrich syndrome;Altman L.C.;Journal of Clinical Investigation,1974
3. Two GTPases, Cdc42 and Rac, bind directly to a protein implicated in the immunodeficiency disorder Wiskott‐Aldrich syndrome;Aspenstrom P.;Current Biology,1996
4. Wiskott‐Aldrich syndrome protein (WASp) is a binding partner for c‐Src family protein‐tyrosine kinases;Banin S.;Current Biology,1996
5. Colony‐stimulating factor‐1 induces rapid behavioural responses in the mouse macrophage cell line, BAC1.2F5;Boocock C.A.;Journal of Cell Science,1989
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