High copy number suppression of the meiotic arrest caused by admc1mutation:REC114imposes an early recombination block andRAD54promotes aDMC1-independent DSB repair pathway-Reference-Cited by-同舟云学术

High copy number suppression of the meiotic arrest caused by admc1mutation:REC114imposes an early recombination block andRAD54promotes aDMC1-independent DSB repair pathway

Published:1999-08 Issue:8 Volume:4 Page:425-444
ISSN:1356-9597
Container-title:Genes to Cells
language:en
Short-container-title:Genes to Cells

Author:

Bishop Douglas K.,Nikolski Yuri,Oshiro June,Chon Jason,Shinohara Miki,Chen Xi

Publisher

Wiley

Subject

Cell Biology,Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1046/j.1365-2443.1999.00273.x/fullpdf

Reference64 articles.

1. Semidominant suppressors of Srs2 helicase mutations of Saccharomyces cerevisiae map in the RAD51 gene, whose sequence predicts a protein with similarities to procaryotic RecA proteins.

2. Sister chromatid-based DNA repair is mediated by RAD54, not by DMC1 or TID1

3. RecA homologs Dmc1 and Rad51 interact to form multiple nuclear complexes prior to meiotic chromosome synapsis

4. DMC1: A meiosis-specific yeast homolog of E. coli recA required for recombination, synaptonemal complex formation, and cell cycle progression

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