Correction of the ΔPhe508 Cystic Fibrosis Transmembrane Conductance Regulator Trafficking Defect by the Bioavailable Compound Glafenine-Reference-Cited by-同舟云学术

Correction of the ΔPhe508 Cystic Fibrosis Transmembrane Conductance Regulator Trafficking Defect by the Bioavailable Compound Glafenine

Published:2010-03-03 Issue:6 Volume:77 Page:922-930
ISSN:0026-895X
Container-title:Molecular Pharmacology
language:en
Short-container-title:Mol Pharmacol

Author:

Robert Renaud,Carlile Graeme W.,Liao Jie,Balghi Haouaria,Lesimple Pierre,Liu Na,Kus Bart,Rotin Daniela,Wilke Martina,de Jonge Hugo R.,Scholte Bob J.,Thomas David Y.,Hanrahan John W.

Publisher

American Society for Pharmacology & Experimental Therapeutics (ASPET)

Subject

Pharmacology,Molecular Medicine

Reference40 articles.

1. Synthesis of novel 4-substituted-7-trifluoromethylquinoline derivatives with nitric oxide releasing properties and their evaluation as analgesic and anti-inflammatory agents

2. Salivary secretion assay for drug efficacy for cystic fibrosis in mice

3. Correctors of Protein Trafficking Defects Identified by a Novel High-Throughput Screening Assay

4. Defective intracellular transport and processing of CFTR is the molecular basis of most cystic fibrosis

5. Altered chloride ion channel kinetics associated with the ΔF508 cystic fibrosis mutation

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