Bypassing Glutamic Acid Decarboxylase 1 (Gad1) Induced Craniofacial Defects with a Photoactivatable Translation Blocker Morpholino-Reference-Cited by-同舟云学术

Bypassing Glutamic Acid Decarboxylase 1 (Gad1) Induced Craniofacial Defects with a Photoactivatable Translation Blocker Morpholino

Published:2018-09-10 Issue:1 Volume:10 Page:266-278
ISSN:1948-7193
Container-title:ACS Chemical Neuroscience
language:en
Short-container-title:ACS Chem. Neurosci.

Author:

O’Connor Matthew J.¹,Beebe Lindsey L.²,Deodato Davide¹,Ball Rebecca E.³,Page A. Tyler³,VanLeuven Ariel J.³,Harris Kyle T.⁴,Park Sungdae²,Hariharan Vani³,Lauderdale James D.³⁵,Dore Timothy M.¹⁴^ORCID

Affiliation:

1. New York University Abu Dhabi, PO Box 129188, Abu Dhabi, United Arab Emirates

2. Department of Genetics, University of Georgia, Athens, Georgia 30602, United States

3. Department of Cellular Biology, University of Georgia, Athens, Georgia 30602, United States

4. Department of Chemistry, University of Georgia, Athens, Georgia 30602 United States

5. Neuroscience Division of the Biomedical and Health Sciences Institute, Athens, Georgia 30602, United States

Funder

Division of Chemistry

National Institute of Neurological Disorders and Stroke

Publisher

American Chemical Society (ACS)

Subject

Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine

Link

http://pubs.acs.org/doi/pdf/10.1021/acschemneuro.8b00231

Reference106 articles.

1. Alteration of epileptogenesis genes

2. Spontaneous Seizures and Altered Gene Expression in GABA Signaling Pathways in a mind bomb Mutant Zebrafish

3. Epilepsy in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase

4. Molecular and cellular mechanisms of altered GAD1/GAD67 expression in schizophrenia and related disorders

5. Decrease in Reelin and Glutamic Acid Decarboxylase67 (GAD67) Expression in Schizophrenia and Bipolar Disorder

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