D-685 Reverses Motor Deficits and Reduces Accumulation of Human α-Synuclein Protein in Two Different Parkinson’s Disease Animal Models
Author:
Affiliation:
1. Department of Pharmaceutical Sciences, Wayne State University, Detroit, Michigan 48202, United States
2. Department of Neurosciences, University of California, San Diego, La Jolla, California 92093, United States
Funder
National Institute of Neurological Disorders and Stroke
Michigan Kick Start award
Publisher
American Chemical Society (ACS)
Subject
Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine
Link
https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.2c00655
Reference71 articles.
1. Parkinson's disease dementia: convergence of α-synuclein, tau and amyloid-β pathologies
2. Slowing of neurodegeneration in Parkinson's disease and Huntington's disease: future therapeutic perspectives
3. Functional Anatomical and Behavioral Consequences of Dopamine Receptor Stimulation
4. MOLECULAR PATHOPHYSIOLOGY OF PARKINSON'S DISEASE
5. Recent advances in the genetics and pathogenesis of Parkinson disease
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